摘要:
[1]Nagata S. Apoptosis by death factor. Cell, 1997;88:355
[2]Zou H, Henzel WJ, Liu X et al. Apaf-1, a human protein homologous to C. Elegans ced-4 participates in cytochrome c-dependent activation of caspase-3. Cell,1997;90(8) : 405
[3]Nicholson DW, Thornberry NA. Caspases: killer proteases. Trands Biochem Sci, 1997;22(8) : 299
[4]Hakem R, Hakem A, Duncan GS et al. Differential requirement for caspase 9 in apoptotic pathways in vivo.Cell, 1998;94(7) : 339
[5]Kuide K, Lippke JA, Ku G et al. Altered cytokine export and apoptosis in mice deficient in interleukin-1 beta coverting enzyme. Science, 1995;267 : 2000
[6]Kuida K, Zheng ZS, Na S et al. Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice. Nature, 996;384 : 368
[7]Li P, Allen H, Banerjee S. Mice deficient in IL-1 β-converting enzyme are defective in production of mature IL1 β and resistant to endotoxic shock. Cell, 1995;80:401
[8]Bergeron L, Perez GI, Macdonald G et al. Defects in regulation of apoptosis in caspase-2-deficient mice.1998 ; (Genes Dev)12 : 1304
[9]Wang S, Miura M, Jung YK et al. Murine caspase-11,an ICE-interacting protease, is essential for the activation of ICE. Cell, 1998;92(4) : 501
[10]Woo M, Hakem R, Soengas MS et al. Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes. Genes Dev, 1998; 12 : 806
[11]Kuida K, Haydar TF, Kuan CY et al. Reduced apoptosis and cytochrome c-mediated caspase activation in mice lacking caspase 9. Cell, 1998;94(7) : 325
[12]Ibrado AM, Huang Y, Fang G et al. Overexpression of Bcl-2 or Bcl-xl inhibits Ara-C-induced CPP32/Yama protease activity and apoptosis of human acute myelogenous leukemia HL-60 cells. Cancer Reseach, 1996;56 : 4743
[13]Hallan E, Blomhoff HK, Smeland EB et al. Involvement of ICE (caspase) family in γ-radiation-inducedapoptosis of normal B lymphocytes. Scand J Immunol,1997;46 : 601 (Continue on P14)
[14]Roberts RA, Nebert DW, Hickman .JA et al. Perturbation of